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published in the Journal of Applied Physiology confirmed that BPC-157 significantly accelerated outgrowth of Achilles tendon explants, markedly increased in vitro tendon fibroblast migration in a dose-dependent manner, and substantially increased fibroblast survival under H₂O₂-induced oxidative stress — establishing a three-part cellular mechanism (outgrowth, migration, survival) that distinguishes BPC-157’s tendon repair profile from direct mitogenic agents