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Its primary distinction is its dual-pathway mechanism (VEGFR2 + NO) combined with its gastric stability — making it the only compound in this group with demonstrated activity when administered orally in animal models, not just by injection
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There was full occlusion/occlusion-like syndrome, additionally presented with intracranial (superior sagittal sinus) hypertension, portal, and caval hypertension, aortal hypotension, progressed venous and arterial thrombosis peripherally and centrally, congested (i.e., inferior caval vein and superior mesenteric vein) and/or failed (i.e., azygos vein) major veins, and ECG disturbances